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. 2019 Sep 13;9:13254. doi: 10.1038/s41598-019-49684-y

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Defects in Fatp4^−/− cornified envelope (CE). The CE was isolated from the dorsal skin of 17.5-day embryos and viewed under a phase contrast microscope. The CE was smaller in Fatp4 mutants (d) compared to controls (a–c) but was normalized by transgene-derived FATP4 (e) and FATP1 (f). Representative images are shown from at least two mice of each genotype. IVL, involucrin promoter.