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. 2019 Sep 16;9:13315. doi: 10.1038/s41598-019-49633-9

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The ced-4(EFH1) mutant exhibits increased axonal regrowth consequent to axotomy. (a) Depiction of measures of axonal regrowth from ALM touch receptor neurons. (b–d) The axonal regrowth measures for the ced-4 null mutant and the engineered EF-hand mutants. Total n numbers are given inside bars. All data come from >3 independent trials, *p ≤ 0.05, **p < 0.01, or ns (not significant) as compared to the ced-4(+) wild type, unpaired two-tail t-test (b,d) or one-way ANOVA with Tukey adjustment for multiple comparison (c); for (d), p = 0.0545.