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. 2019 Sep 16;9:13315. doi: 10.1038/s41598-019-49633-9

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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A ced-3 mutant allele that disrupts the caspase active site suppresses the enhanced axonal regrowth of the ced-4(EFH1) mutant. (a) ced-3(n2433), lacking the caspase active site (indicated ced-3(lf)), exhibits reduced regeneration. Data come from >3 independent trials, *p < 0.05, unpaired t-test. (b) impact of ced-4(EFH1) on regeneration outcomes. Data come from >3 independent trials, **p < 0.01 or ****p < 0.0001, one-way ANOVA with Tukey adjustment for multiple comparison; numbers of animals scored indicated in bars. Separate graphs are given for time-separated trials.