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. 2019 Sep 17;9:13445. doi: 10.1038/s41598-019-49937-w

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Increased GM-CSF and granulopoiesis were detected in hematopoietic disorder patients with EBV infection. (A) Comparison of granulocyte population in BM between EBV-infected and uninfected patients. Anti-human CD66b Ab was used for the detection. Collected EBV⁺ patient’s samples were CAEBV (n = 1), NKTL (n = 2), and EBV^- patient’s samples were DLBCL (n = 1), FL (n = 2) patients, respectively. Formal name of each abbreviations is bellow; CAEBV = Chronic active EBV infection, NKTL = NK/T-cell lymphoma, DLBCL = Diffuse large B-cell lymphoma, FL = Follicular lymphoma. Representative data are indicated. (B) ELISA assay of hGM-CSF in serum of patients. EBV⁺ data were detected from HLH (N = 8) and PTLD (N = 4) patients, EBV⁻ data were detected from sJIA (N = 2) and KD (N = 4) patients. Data are mean ± SD. Significant difference was detected between EBV infected patients and uninfected patients. HLH = Hemophagocytic lymphohistiocytosis, PTLD = post-transplantation lymphoproliferative disorder, sJIA = systemic juvenile idiopathic arthritis, KD = Kawasaki disease. *p < 0.05.