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. 2019 Sep 17;9:13443. doi: 10.1038/s41598-019-49617-9

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Lack of differences in PBMC mRNA expression levels of OXTR and AVPR1A compared in ASD children, their neurotypical siblings, and neurotypical age-matched children. Findings are shown for our real-time PCR findings (a,b). Corroborating our findings, no differences between ASD and control children were found in Gene Expression Omnibus (GEO) datasets from PBMC (c–f) or whole blood (g–j) samples analyzed by datamining for their OXTR and AVPR1A expression. GSE codes and cohort sizes are shown. Note the large distribution of expression levels for OXTR and AVPR1A in both our PBMC samples (a,b) and the GEO datasets (c–j). Between-group differences were analyzed by one-way ANOVA test (a,b) and by Student’s t-test (c–j).