(
a) Defective Kip3 is synthetic lethal with
dyn1∆, whereas defective Kip2 or Bik1 is synthetic lethal with
bim1∆. Here we show that the expression of the Kip3-3xsfGFP, Kip2-3xsfGFP, and Bik1-3xGFP proteins does not cause synthetic growth defect or lethality with
dyn1∆ or bim1∆. Spot growth assay of background cells (Spc72-GFP) and cells of indicated genotype for two days on solid YPD agar at 30°C. Spots are from exponentially growing cell samples that were sequentially diluted five-fold. (
b, c) Scatterplots with fitted linear regression lines (black) between two-dimensional (2D) b-microtubule length and normalized Kip3-3xsfGFP (
b) or Kip2-3xsfGFP (
c) intensity on b-microtubule plus-ends (%). Kip3-3xsfGFP shows a significant positive correlation (p<0.0001, n = 183 cells) in contrast to Kip2-3xsfGFP, which shows no significant correlation (p=0.3, n = 200 cells). In addition, Kip3-3xsfGFP is nearly absent on very short microtubules, whereas Kip2-3xsfGFP is equally abundant on microtubules of all lengths. (
d) Technical replicates for the quantifications of fluorescence intensities (a.u.) from endogenous Kip2-3xsfGFP along preanaphase b-microtubules. The signals were processed and presented as in
Figure 1, and fluorescence intensities were normalized (see Materials and methods) to the data shown in
Figure 1. Left panel: 15 ≤ n ≤ 69 per bin, right panel: 47 ≤ n ≤ 98 per bin.