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. 2019 Aug 1;20(4):3027–3034. doi: 10.3892/mmr.2019.10547

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Extent of AP-induced damage in the Sham, AP and CUR groups. (A) Ascites volume in each group 12, 24 and 36 h after surgery. (B) ABG level (PaO₂ and PaCO₂) in each group. (C) Amylase activity in each group at 12, 24 and 36 h after surgery. (D) Weight of the pancreas in each group at 12, 24 and 36 h after surgery. (E) Hematoxylin and eosin staining of pancreatic tissue in each group at ×100 and ×200 magnification. *P<0.05, **P<0.01. Sham, control; AP, acute pancreatitis; CUR, curcumin; ABG, arterial blood gas; Pa, partial pressure.