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. 2019 Aug 9;20(4):3240–3248. doi: 10.3892/mmr.2019.10585

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Copyright: © Gong et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — KLF4 overexpression inhibits the mTOR signaling pathway in podocytes treated with DKD mouse serum. (A) Representative bands of mTOR, S6K, p-mTOR and p-S6K expression in the individual groups. (B) Bar graphs showing the quantitative evaluation of the level of mTOR/β-actin, p-mTOR/mTOR, S6K/β-actin, and p-S6K/S6K in the individual groups. Data are presented as the mean ± SD (n=5). *P<0.05; **P<0.01. KLF4, Krüppel-like factor 4; p, phosphorylated; DKD, diabetic kidney disease; NC, negative control.