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. Author manuscript; available in PMC: 2019 Sep 24.
Published in final edited form as: Basic Res Cardiol. 2016 Dec 19;112(1):5. doi: 10.1007/s00395-016-0594-x

Fig. 5.

Fig. 5

NO at a concentration subthreshold for dilation suppresses FMD in atrial arterioles from patients with and without CAD. a FMD was abolished by incubation with NONOate in atrial arterioles from patients with but not without CAD. b, c Treatment of adipose arterioles with L-NAME for either 1 or 18 h shifted the mechanism of dilation from NO to H2O2 (Peg-catalase inhibitable). The dilation was abolished by treatment with DETANONOate. *P < 0.05 two-way ANOVA RM Tukey’s post hoc analysis, N = 3–6