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Probe-set signal intensities of significantly up-regulated DRG genes in network Cluster B in response to tail amputation involved in the mediation of inflammatory and neuropathic pain signaling. (A) KCNK3, Potassium two pore domain channel subfamily K member 3 and neuronal high affinity glutamate transporter; (B) SLC1A1, Solute carrier family 1 member 1. All signal intensity data values are shown by treatment group (open circles, n = 95) along with group means ± SEM.
