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. 2019 Sep 18;6:314. doi: 10.3389/fvets.2019.00314

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Copyright © 2019 Sandercock, Barnett, Coe, Downing, Nirmal, Di Giminiani, Edwards and Freeman.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Probe-set signal intensities of selected significantly down-regulated DRG voltage-gated potassium channel genes after tail amputation in network Cluster A associated with inflammatory and neuropathic pain signaling. (A) KCNA4, Potassium voltage-gated channel subfamily A member 4; (B) KCNG4, Potassium voltage-gated channel modifier subfamily G Member 4; (C) KCNQ3, Potassium voltage-gated channel subfamily Q member 3; (D) KCNH6, Potassium voltage-gated channel subfamily H Member 6; (E) KCNV1, Potassium voltage-gated channel modifier subfamily V member 1; (F) KCNT2, Potassium sodium-activated channel subfamily T member 2; (G) KCNK1, Potassium two pore domain channel subfamily K member 1; (H) KCNK10, Potassium two pore domain channel subfamily K member 10. All signal intensity data values are shown by treatment group (open circles, n = 95) along with group mean ± SEM.