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. 2019 Sep 18;6:314. doi: 10.3389/fvets.2019.00314

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Copyright © 2019 Sandercock, Barnett, Coe, Downing, Nirmal, Di Giminiani, Edwards and Freeman.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Probe-set signal intensities of DRG genes in network Cluster B involved in inflammatory and neuropathic pain pathways expressing significant up-regulated transcript activity (3-way ANOVA, p < 0.05) 1, 8, and 16 weeks after tail amputation at two ages (3 and 63 days). Genes shown are linked to regulation of gene expression, inflammation, proteinase inhibition, and neurotropic signaling. (A) ATF3, Activating transcription factor 3; (B) JUN, Jun proto-oncogene, AP-1 transcription factor subunit; (C) PLA2G3, Phospholipase A2 group III; (D) PTGS2, Prostaglandin-endoperoxide synthase 2; (E) SERPINB2, Serpin family B member 2; (F) TIMP1, TIMP metallopeptidase inhibitor 1. All signal intensity data values are shown by treatment group (open circles, n = 95) along with group means ± SEM.