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. 2019 Sep 24;9:13726. doi: 10.1038/s41598-019-49396-3

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Overview of the non-canonical and coupled with digestion transcriptional regulation of heme oxygenase in Aedes aegypti midgut. Schematic representation of a female of A. aegypti mosquito after a blood meal and in detail, the inside of a midgut cell – amino acids activate target-of-rapamycin (TOR) pathway, which induces the transcription of AeHO (presumably through the GATA factor) resulting in heme degradation and production of AeBV, thus contributing to gut homeostasis and hematophagic adaptation.