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. 2018 Dec 14;26(7):234–247. doi: 10.1038/s41417-018-0067-5

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — SNHG14 was upregulated following I/R in vitro and in vivo. a Representative brain sections of focal ischemia induced by MCAO were exhibited by TTC staining. Quantitative analysis of brain infarct volume after I/R in rats were shown by bar graph. b The expression of SNHG14 level was detected by qRT-PCR at 12, 24, and 48 h following MCAO/R in rats. c Representative images exhibiting the expression level and intracellular localization of SNHG14 were determined by RNA-FISH at 12, 24, and 48 h following MCAO/R in rats. a–c N = 6 per group. d The expression level of SNHG14 was examined by qRT-PCR in PC-12 cells after OGD/R. e Representative images exhibiting the expression level and intracellular localization of SNHG14 were determined by RNA-FISH in PC-12 cells after OGD/R. Data were expressed as mean ± SD. ***P < 0.001 represents statistically difference. Scale bar = 100 μm