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. 2019 Jul 11;33(10):1303–1316. doi: 10.1177/0269881119856850

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© The Author(s) 2019

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 4. — Tissue glucose utilization during hyperinsulinemic euglycemic clamp in female rats. (a) OLZ significantly decreased muscle glucose utilization (combined gastrocnemius, extensor digitorum longus and soleus muscle, *p < 0.05). Co-administration of SAM appeared to attenuate this effect. (b) OLZ significantly increased glucose utilization in adipose tissue (combined inguinal and retroperitoneal adipose tissue, *p < 0.05), which was blocked by co-administration with SAM (#p < 0.001). Data are expressed as mean ± SEM.

OLZ: olanzapine; SAM: samidorphan; VEH: vehicle