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. 2018 Aug 23;15(5):1163–1175. doi: 10.5114/aoms.2018.77733

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Copyright: © 2018 Termedia & Banach

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International (CC BY-NC-SA 4.0) License, allowing third parties to copy and redistribute the material in any medium or format and to remix, transform, and build upon the material, provided the original work is properly cited and states its license.

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SMYD3 promoted cell proliferation in MEC1 and CCL cancerous cells through increasing STAT3 expression. Cells were plated into multi-well plates and divided into four groups to apply four different treatments: NC group, negative control, STAT3 knockdown group, indicating transfection with siRNA1-STAT3, SMYD3 overexpression group, indicating transfection with pcDNA3.1-SMYD3 vectors and combination group, indicating transfection with siRNA-STAT3 and pcDNA3.1-SMYD3. A – Cell viability was detected in MEC1 cell line by MTT assay. B – Cell viability was detected in CCL cell line by MTT assay. C – Cell viability was detected in MEC1 cell line by MTT assay. D – Cell viability was detected in CCL cell line by MTT assay (B)