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. 2019 Sep 27;10:4422. doi: 10.1038/s41467-019-12267-6

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Scheme for study design and screening results. First, we conducted discovery GWAS followed by imputation using 1000 Genomes Project Phase1 as a reference. Of the 2997 SNPs that were associated with PCa (P < 1 × 10⁻⁵), top 101 SNPs excluding those residing in previously reported loci were evaluated in the replication study using an independent cohort. A total of 12 SNPs were significantly associated with PCa (P < 5 × 10⁻⁸) after meta-analysis of discovery and replication cohorts