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. 2019 Sep 27;10:4033. doi: 10.1038/s41467-019-12052-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Positional cloning of Rsv4. a Fine mapping of Rsv4. The gene structures in the Peking, Williams 82, and Enrei genomes are shown at the bottom. Indels are indicated in blue. Black boxes are untranslated regions. b Predicted Rsv4 protein structure. Amino acid positions of the transmembrane (TM) helix and active-site residues of the RNase H family domain are shown