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. 2019 Sep 9;116(39):19743–19752. doi: 10.1073/pnas.1900317116

Fig. 6.

Fig. 6.

Opposing effects of Bcpg1 and Bcpme1/2 mutations in virulence. (A) Extracted ion chromatograms obtained by HP-SEC-MS analysis in negative ionization mode of OGs endogenously produced during infection of A. thaliana leaves infected by WT B. cinerea strain and Bcpg1, Bcpme1/2 mutants over time. (B and C) Infection assays of WT B. cinerea and mutant strains on 5-wk-old (B) or 6-wk-old (C) rosette leaves of A. thaliana. Statistical data of lesion size 72 hpi. Values are means ± SEM (n = 15); **P < 0.01 and ***P < 0.001 by Student test. (D) B. cinerea coinfection assay on rosette leaves of A. thaliana. Leaves were infected with WT B. cinerea strain or Bcpme1/2 followed 24 h later by Bcpg1 or a mock control (PDB medium). Lesion size of the infected leaves were measured 72 h later. Values are means ± SEM (n = 25); *P < 0.05 by Student test; ns: nonsignificant.