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. Author manuscript; available in PMC: 2020 Nov 1.
Published in final edited form as: J Urol. 2019 Oct 9;202(5):880–889. doi: 10.1097/JU.0000000000000257

Table 2:

Summary of Biomarker Studies for Overactive Bladder (OAB)

Biomarker # Studies Summary of Effect
NGF 40 Multiple poorer quality studies showing associations between urinary NGF and OAB; higher quality studies do not show differences between NGF and controls when controlling for covariates, especially age. Limited data showing that serum NGF may be elevated in a subset of patients with OAB and metabolic syndrome, but not in controls or other types of OAB. Two studies in bladder tissue fail to demonstrate differences in urothelial NGF between OAB and controls.
BDNF 7 Multiple poorer quality studies showing associations between BDNF and OAB; higher quality studies do not show differences after controlling for potentially confounding factors
PGE2 7 Mixture of effect; 4 poor quality studies showing an association while 3 poor quality studies do not
ATP 6 Variable results in 3 studies; 3 additional studies performed using urodynamic fluid (not solely in urine)
CRP 13 Conflicting results are noted in multiple analyses using regression to control for potentially confounding variables.
Other inflammatory 17 Overall poor quality studies that do not control for confounders and often do not adjust for multiple testing; variable associations noted (see Supplementary Tables 2,5)
Receptors/Channels 11 Multiple studies showing higher M3 muscarinic receptor expression/density and lower β−3 adrenergic receptor expression in OAB; no differences in M2 receptors between OAB and controls. More TRPV1 receptors in OAB; variable results with regards to purinergic receptors and gene for beta 3 adrenoreceptor (ADR3B)
Gene expression/proteomic 2 Small sample sizes, lack of adjustment for confounders, and lack of adjustment for false-discovery rate limit conclusive results
Metabolomic 2 Metabolites may be useful in phenotyping (understanding subtypes of OAB)
Microbiome 9 Mostly association studies; require further investigation with longitudinal sampling and studies that control for potentially confounding factors

NGF: nerve growth factor, BDNF: brain derived neurotrophic factor; PGE2: prostaglandin E2; ATP: adenoside triphosphate; CRP: C-reactive protein

Individual study details are listed in Supplemental Table 2