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. 2019 Jun 27;8(10):1068–1083. doi: 10.1002/sctm.19-0013

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© 2019 The Authors. stem cells translational medicine published by Wiley Periodicals, Inc. on behalf of AlphaMed Press

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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Angiogenesis evaluated by α‐SMA and CD31 staining. (A): Representative images of α‐SMA and CD31 staining in peri‐infarcted region at the endpoint in each group. Vessels formation was stained green and nuclei were dyed with DAPI (blue). Scale bar = 25 μm. (B): Quantitative analyses of CD31 and (C) α‐SMA staining with vessel density per HPF in each group. n = 5 for each group. ^$ p < .05 vs. Sham group; ^& p < .05 vs. AMI group; ^γ p < .05 vs. Early1; ^κ p < .05 vs. Early2; ^φ p < .05 vs. Early3; ^α p < .05 vs. Mid1; ^μ p < .05 vs. Mid2; ^# p < .05 vs. Mid3; *p < .05 intragroup comparison in different periods. All data are expressed as mean ± SD. Abbreviations: α‐SMA, α‐smooth muscle actin; AMI, acute myocardial infarction.