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. 2019 Sep 17;20(18):4582. doi: 10.3390/ijms20184582

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© 2019 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Uptake of apoE3/rHDL and acro-apoE3/rHDL by bEnd.3 cells. Uptake was followed by direct fluorescence in the presence of excess LDL (A) or suramin (B). Uptake experiments were carried out in the presence of 100× excess LDL or 2mM suramin and apoE3/rHDL (a–c) or acro-apoE3/rHDL (d–f) (apoE3 concentration: 3 µg/mL). The panels show fluorescence of DAPI (a,d), DiI (b,e), and Merge (c,f).

Uptake of apoE3/rHDL and acro-apoE3/rHDL by bEnd.3 cells. Uptake was followed by direct fluorescence in the presence of excess LDL (A) or suramin (B). Uptake experiments were carried out in the presence of 100× excess LDL or 2mM suramin and apoE3/rHDL (a–c) or acro-apoE3/rHDL (d–f) (apoE3 concentration: 3 µg/mL). The panels show fluorescence of DAPI (a,d), DiI (b,e), and Merge (c,f).