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. 2000 Jun 15;20(12):4506–4514. doi: 10.1523/JNEUROSCI.20-12-04506.2000

Fig. 2.

Fig. 2.

Increased phosphorylation of JNK, p38, and ERK after 5 min of transient forebrain ischemia. A, Western immunoblots of hippocampal homogenates of sham-operated gerbils (lane 1) and postischemic gerbils at 0 min (lane 2) and 30 min (lane 3) after 5 min of transient forebrain ischemia (top panel, active JNK; middle panel, active p38; bottom panel, active ERK;p-, phosphorylated). B, Quantitative representation of immunoblots showing activation of JNK1 and p38 30 min after ischemia and ERK2 immediately after ischemia in the hippocampal extracts. The data presented are representative of at least three separate experiments.