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. 2019 Oct 2;9:14151. doi: 10.1038/s41598-019-50478-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The role of the width of the infarcted area with a fixed length of 25% and a fixed position of 0% in excitation wave propagation in the circular ring tissue. (a) Structural representation and wave conduction tested with two stimuli with S1-S2 intervals of 520 ms in the 2D ring model of infarcted tissue without fibrosis at different times. (b) Structural representation and wave conduction tested with two stimuli with S1-S2 intervals of 520 ms in the 2D ring model of infarcted tissue with 20% fibrosis at different times. (c) Change in the size of the vulnerable windows in the circular ring tissue with and without 20% fibrosis as a function of the width of the infarcted tissue. Black bars: infarcted tissue without fibrosis; Red bars: infarcted tissue with 20% random fibrosis.