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. 2019 Oct 2;9:14151. doi: 10.1038/s41598-019-50478-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Wave propagation in the 2D, actual ventricular tissue and ideal circular ring tissue models. (a) Structural representation and wave propagation tested with two stimuli with S1-S2 intervals of 520 ms in the 2D actual ventricular tissue model of patient 1 and in the corresponding ideal ring strip tissue model with and without 20% fibrosis. (b) Structural representation and wave propagation tested with two stimuli with S1-S2 intervals of 520 ms in the 2D actual ventricular tissue model of patient 3 and in the corresponding ideal ring strip tissue model with and without 20% fibrosis. (c) The vulnerable windows of the actual infarcted tissues of patient 1 and patient 3 and their corresponding ring strip tissues with and without 20% fibrosis. Black bars: tissue without fibrosis; Red bars: tissue with 20% random fibrosis.