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. Author manuscript; available in PMC: 2019 Oct 3.
Published in final edited form as: Basic Res Cardiol. 2018 Mar 9;113(3):16. doi: 10.1007/s00395-018-0674-1

Fig. 4.

Fig. 4

a Protocol 3A: effect of Krebs–Henseleit (KH) buffer, serum and serum fractions (supernatant-bottom, supernatant-top), obtained from Zucker lean rats following the standard RIPC stimulus, on % cell death (mean ± SEM) of HL-1 cells subjected to 2.5 h of hypoxia and 24 h of reoxygenation. *p < 0.05 versus KH buffer. b Protocol 3B: effect of KH buffer, serum and serum fractions (supernatant-bottom, supernatant-top), obtained from Zucker fatty rats following the standard RIPC stimulus, on % cell death (mean ± SEM) of HL-1 cells subjected to 2.5 h of hypoxia and 24 h of reoxygenation. *p < 0.05 versus KH buffer