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. 2019 Mar 7;44(10):2314–2324. doi: 10.1007/s11064-019-02755-y

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 4 — Common regulator analysis of hypothalamic preoptic areal proteins. Abbreviations: Transcription factor E2f (E2f), Transcription factor AP-1/Proto-oncogene c-Fos (Jun/Fos), Platelet-derived growth factor (Pdgf), Phosphatidylinositol-3-kinases (Pi3k), Rac-alpha serine/threonine-protein kinase (Akt1), Serine/threonine-protein kinase mTOR (Mtor), Peptidyl-prolyl cis–trans isomerase NIMA-interacting 1 (Pin1), Thioredoxin (Txn), Heat shock factor protein 1 (Hsf1), Cyclic AMP-responsive element-binding protein 1 (Creb1), Early growth response protein 1 (Egr1), Myc proto-oncogene protein (Myc), Hypoxia-inducible factor 1-alpha (Hif1a), Transcription factor Sp1 (Sp1), Signal transducer and activator of transcription 3 (Stat3), Cellular tumor antigen p53 (Tp53), Insulin (Ins), Transforming growth factor beta-1 (Tgfb1). Edges indicate the relationships between common regulators and altered maternal proteins. Red or blue highlights indicate maternal proteins that were significantly increased or decreased. Full protein names are presented in Fig. 6. (Color figure online)