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. 2019 Aug 26;18(5):3397–3404. doi: 10.3892/etm.2019.7939

Table I.

Primers used in the current study.

Gene Primer sequence (5′-3′)
Bcl-2 F: 5′-CACAAGAGGCCAAGGCTACC-3′
R: 5′-CAGGAAAGCAGGAAGTCTCAA-3′
Bcl-xl F: 5′-CCAAAATCCCTGCTCTTCATG-3′
R: 5′-GCATTCTTGGCATCGTTATTCA-3′
Bax F: 5′-ATTGAGAAACGATTTGCCTAC-3′
R: 5′-GGAAATGGCTTATTCTCCTTTGCTT-3′
Bad F: 5′-GGAAACCCGGTGGGGCCAC-3′
R: 5′-ACCAGTAGCGGGTGGTC-3′
Iba-1 F: 5′-CACCATTAGCTGGGCGTCT-3′
R: 5′-GATGCGGAAGTAGCAAAAGC-3′
MPO F: 5′-ATTCTCCACACCCTGTTTCG-3′
R: 5′-ATGCAGCAGTGTGTCATTCC-3′
TGF-β F: 5′-GGCCAGATCCTGTCCAAGC-3′
R: 5′-GTGGGTTTCCACCATTAGCAC-3′
ERK F: 5′-ACCTAGCCGTGGAGCTTGG-3′
R: 5′-GCCCTTGGTTGTTTACCTGG-3′
β-actin F: 5′-AAGGACCTGTATGCCAACACA-3′
R: 5′-ATCCACACAGAATACTTGCGTT-3′

Bcl-2, B cell lymphoma-2; Bcl-xl, B cell lymphoma-extra large; Bax, Bcl-2-assocaited X protein; Bad, Bcl-2-associated death promoter; Iba-1, ionized calcium binding adaptor molecule 1; MPO, myeloperoxidase; TGF-β; transforming growth factor-β; ERK, extracellular signal-regulated kinase.