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. 2019 Aug 26;18(5):3534–3542. doi: 10.3892/etm.2019.7942

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Copyright: © Duan et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Knockdown of CCEPR inhibits EMT in colorectal cancer cells. MMP-2 and MMP-9 protein expression was significantly decreased following CCEPR knockdown in (A) HT29 and (B) SW480 cell lines. E-cadherin protein expression was significantly increased, while N-cadherin and vimentin protein expression was significantly decreased, following CCEPR knockdown in (C) HT29 and (D) SW480 cell lines. **P<0.01 vs. siNC. CCEPR, cervical carcinoma expressed PCNA regulatory lncRNA; lncRNA, long non-coding RNA; EMT, epithelial-mesenchymal transition; MMP, matrix metalloproteinase; si, small interfering RNA; NC, negative control.