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. 2019 Oct 7;14:43. doi: 10.1186/s13020-019-0263-8

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Effects of TKSE on Akt, ERK, and AMPK signaling in HT-29 cells. a HT-29 cells were treated with the indicated concentrations of TKSE for 6 h, and the protein levels of Akt and its downstream proteins (mTOR, p70S6K1, and 4E-BP1), ERK, AMPK, and their phosphorylated forms were analyzed by western blot. b Suppression of the effect of TKSE on mitochondrial membrane potential by compound C. ***p < 0.001