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. 2019 Aug 27;18(5):4726–4734. doi: 10.3892/ol.2019.10782

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Copyright: © Chen et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Knockdown of SNHG7 inhibited thyroid cancer cell proliferation and cell cycle. (A) The mRNA expression of SNHG7 was significant decreased by siSNHG7 in CAL62 cells. (B) Knockdown of SNHG7 inhibited cell proliferation compared with control cells in CAL62 cells. (C) The mRNA expression of SNHG7 was significant decreased by siSNHG7 in SW579 cells. (D) Knockdown of SNHG7 inhibited cell proliferation compared with control cells in SW579 cells. (E) Knockdown of SNHG7 in CAL62 cells increased the percentage of cells in the G₁ phase and decreased the percentage of cells in S and G₂ phases compared with control cells. (F) Statistical histogram of cell cycle distribution after SNHG7 silencing in CAL62. (G) Knockdown of SNHG7 in SW579 cells increased the percentage of cells in G₁ phase and decreased the percentage of cells in S phase compared with control cells. (H) Statistical histogram of cell cycle distribution after SNHG7 silencing in SW579 cells. *P<0.05; **P<0.01; ***P<0.001, vs. siNC. SNHG7, small nucleolar RNA host gene 7; si, small interfering; NC, negative control.