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. 2019 Sep 13;18(5):5043–5054. doi: 10.3892/ol.2019.10866

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Copyright: © Bian et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 7. — Gene set enrichment analysis of CEACAM7. Two significantly enriched pathways were identified in the low CEACAM7 expression group of patients with CRC at nominal P<0.05 and FDR<0.25, including (A) glycosaminoglycan biosynthesis-chondroitin sulfate (nominal P=0.002; FDR, 0.101) and (B) ECM receptor interaction (nominal P=0.022; FDR, 0.151). CEACAM7, carcinoembryonic antigen-related cell adhesion molecule 7; CRC, colorectal cancer; NES, normalized enrichment score; FDR, false discovery rate; ECM, extracellular matrix.