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. 2019 Sep 10;18(5):4691–4702. doi: 10.3892/ol.2019.10846

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Correlations between transcription levels of BSG, STAT3, ITK and PPIA in each cancer type generated from TIMER. As shown in (A-D) and (E-H), BSG and STAT3 correlated with PPIA in each cancer type, with a (A-D) positive correlation for BSG, and a (E-H) negative correlation for STAT3. As shown in (J-L) and (I), the expression levels of ITK were negatively correlated with PPIA in (K) LIHC, (J) LUAD and (L) SKCM, but not (I) LGG. LGG, brain lower grade glioma; LIHC, liver hepatocellular carcinoma; STAT3, signal transducer and activator of transcription 3; TIMER, Tumor IMmune Estimation Resource; BSG, basigin; PPIA, peptidyl-prolyl cis-trans isomerase A; ITK, interleukin-2 tyrosine kinase; LUAD, lung adenocarcinoma; SKCM, skin cutaneous melanoma.