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. 2019 Sep 18;18(5):3767–3774. doi: 10.3892/etm.2019.8018

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Copyright: © Dong et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — β-asarone increases ERK phosphorylation in the hippocampus of chronic unpredictable mild stress-treated rats. (A) Following treatment, the protein expression levels of ERK and p-ERK in different groups were detected by western blot analysis. (B) Quantified protein levels of p-ERK. Data were analyzed and expressed as the mean ± standard deviation. **P<0.01 vs. control group; #P<0.05 and ##P<0.01, vs. model group. β-asarone1/2/3, depression model rats treated with 12.5, 25 or 50 mg/kg β-asarone, respectively; FLU, fluoxetine-treated depression rats; ERK, extracellular signal-regulated kinase; p-ERK, phosphorylated ERK.