Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2019 Sep 16;11(18):7473–7491. doi: 10.18632/aging.102254

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2019 Chen et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

PMC Copyright notice

hnRNPR promoted cell proliferation, migration and invasion of gastric cancer cells. (A) The efficiency of hnRNPR knockdown or overexpression was detected by western blot in the indicated cells after transfection with shhnRNPR or plasmids. GAPDH serve as the internal control. (B) CCK8 and (C) colony formation assays showed that hnRNPR knockdown suppressed cell growth in MGC-803 and AGS, while overexpression of hnRNPR promoted cell proliferation in HGC-27 and SGC-7901. (D) Migration assays and (E) invasion assays revealed that hnRNPR inhibition decreased cell migration and invasion abilities of MGC-803 and AGS, and hnRNPR overexpression increased cell migration and invasion abilities in HGC-27 and SGC-7901. Each experiment was performed in triplicate and repeated three times. P values were calculated with two-tailed unpaired Student’s t test. *, P<0.05, **, P<0.01.