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. 1999 Oct 15;19(20):8740–8746. doi: 10.1523/JNEUROSCI.19-20-08740.1999

Table 1.

Genetic complementation of mutant and wild-typeamn alleles for olfactory memory

Genotype Initial learning1-a 3 hr memory1-b
Mean ± SEM p Mean ± SEM p
+/+1-c 87  ± 2 37  ± 3
amn1/+ 81  ± 21-d 0.294 36  ± 71-d 0.844
amn28A/+ 71  ± 41-d 0.004 28  ± 51-d 0.138
amnX8/+ 80  ± 31-d 0.228 39  ± 41-d 0.730
amn1/amn1 59  ± 41-d <0.001 16  ± 21-d <0.001
amn28A/amn28A 55  ± 31-d <0.001 20  ± 41-d 0.003
amnX8/amnX8 48  ± 41-d <0.001 17  ± 41-d <0.001
amn1/amn28A 67  ± 41-e 0.126 21  ± 41-e 0.443
amn1/amnX8 62  ± 41-e 0.608 20  ± 41-e 0.496
amn28A/amnX8 53  ± 41-e 0.218 18  ± 51-e 0.688
F1-a

n = 6 PIs per group.

F1-b

n = 8 PIs per group.

F1-c

This amn+ allele, which was carried in a w (CS10) stock, is aw1118 allele that was out-crossed for 10 generations to a wild-type Can-S stock and shows normal olfactory learning/memory, olfactory acuity, and shock reactivity (Dura et al., 1993).

F1-d

Planned pairwise comparison (α′ = 0.006) to +/+ after a one-way ANOVA, with genotype as a main effect. Data from initial learning and memory were analyzed separately.

F1-e

Planned pairwise comparison (α′ = 0.006) toamn1/amn1after a one-way ANOVA, with genotype as a main effect. Data from initial learning and memory were analyzed separately.