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Plausible pathophysiological mechanisms linking heat exposure to myocardial infarction.
Heat exposure may lead to the onset of myocardial infarction through:
– Vasodilation and increased surface blood circulation, which may increase cardiac work load and result in hemoconcentration and thrombosis promotion
– Release of interleukins that cause local and systemic inflammation and result in endothelial dysfunction (25, 26).
↑, Increasing; ↓, decreasing
