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Inhibitory effects of the three promiscuous TAS2R agonists on the LPS-induced production of pro-inflammatory cytokines (TNF-α, CCL3, and CXCL8). Lung macrophages from 6 to 7 patients were stimulated with LPS (10 ng.mL–1) in the absence or presence of the TAS2R agonists (1 mM). ∗p < 0.05, ∗∗p < 0.01 compared with LPS alone.
