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. 2019 Mar 11;40(9):1168–1183. doi: 10.1038/s41401-018-0197-1

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — Effects of CSA on the number of immune cells in the infarcted heart and MLNs in prolonged myocardial I/R. a Comparison of the changes in the infarcted heart. The Y-axis shows the ratio (%) of cells of interest to CD45⁺ cells from the heart of the same mouse subjected to flow cytometry analysis (n = 5–7). b Comparison of the changes in MLNs. The Y-axis (except in the first graph) shows the percentage of the chosen cell population among 1.5 × 10⁵ cells from mouse MLNs analyzed by flow cytometry (n = 6). VEH vehicle (control group), CSA cyclosporine A (treatment group). Data are shown as the mean ± SE; n.s. no significance, *P < 0.05 as indicated