Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2019 Oct 10;9:14515. doi: 10.1038/s41598-019-51078-z

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

PMC Copyright notice

Electrophysiological analysis of the sarcolemnic properties of control and eccentric contraction (EC)-damaged skeletal muscle fibers. (A) Resting membrane potential (RMP) recorded in the current clamp mode. (B–D) Membrane passive properties of the myofibers measured in voltage-clamp mode: linear capacitance, C_m, membrane resistance, R_m, and cell conductance, G_m. Data are reported as means ± SEM. *P < 0.05, **P < 0.01 versus control. (E) K⁺ current (I_K) measured in voltage-clamp mode normalized to cell capacitance C_m (pA/pF). The I_K values (means ± SEM) recorded in EC muscles, from −50 mV to 60 mV, were statistically different from those recorded in control muscles (P < 0.05). All measurements were performed on at least 15 myofibers from each of 5 control and 5 EC-injured muscles.