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. 2019 Oct 4;13:1049. doi: 10.3389/fnins.2019.01049

FIGURE 2.

FIGURE 2

Influence of specific blockade of A1 or A2a receptors in the rVLM on EA modulation of GD-induced pressor reflexes. Bars represent increases in mean arterial blood pressure (MAP) following GD. Values below each bar indicate the baseline MAP (means ± SE) before GD. (A,B) Microinjection of DPCPX (3 mM in 50 nl; an A1 receptor antagonist; A) or SCH 58261 (1 mM in 50 nl; an A2a receptor antagonist; B) into the rVLM with EA treatment. ∗P < 0.05, a decrease of GD response after the onset of EA; #P < 0.05, after vs. before microinjection into the rVLM. Labels a–d in (A) and e–h in (B) indicate examples of original BP tracings of a rat from each group; ↓, time of GD application. These data suggest that adenosine A2a, but not A1 receptors in the rVLM contribute to EA modulation of GD-induced pressor reflexes.