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. 2019 Sep 10;18(21):2849–2859. doi: 10.1080/15384101.2019.1638190

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© 2019 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited, and is not altered, transformed, or built upon in any way.

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Figure 6. — Down-regulation of MALAT1 abolished the neuroprotective effect of HA-introduced vitamin B1 and B12 in cerebral palsy rats. Rats were randomly allocated into several groups (n = 6 in each group): Sham, CP, CP+HA-VitB, CP+HA+si-control, and CP+HA+si-MALAT1. (a) The motor and memory functions of rats were evaluated by Rota-rod test and Step-down avoidance task, respectively; the NSE level was detected by ELISA. (b) The expression of MALAT1, miR-1, and BDNF mRNA in hippocampus tissues was quantified with qRT-PCR; the level of BDNF protein in hippocampus tissues was analyzed by western blot. *P < 0.05 compared with sham; ^#P < 0.05 compared with CP.