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. 2019 Oct 14;9:14746. doi: 10.1038/s41598-019-49327-2

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Loss of OMA1 alters mitochondrial network organization and actin branching in 21MT-1 cells. (A) Visualization of mitochondria in 21MT-1 and OM.21 cells. Image of MitoTrackerR Red CMXRos-stained mitochondria in each respective cell type were captured using confocal fluorescent microscopy, Scale bar, 5 μm. (B) Confocal fluorescent images of immunostained actin fibers in paraformaldehyde-fixed control and OM.21 cells show actin branching in the OMA1-depleted breast cancer cells, Scale bar, 5 μm.