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. 2019 Oct 14;10(10):777. doi: 10.1038/s41419-019-2020-4

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — The levels of COX2 and MET were shown in HCC827GR cells a, HCC827GR cells with MET/COX2 gene silencing b, c, and HCC827 cells stably expressing COX2 d by western blotting analysis. e, f IHC analysis of COX2 and MET expression in TMA. Data are representative of results from triplicate experiments. Magnifications, ×40, ×100, ×200