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. 1997 Jan 1;17(1):204–215. doi: 10.1523/JNEUROSCI.17-01-00204.1997

Table 1.

Functional properties of AMPARs in neurons of the basal ganglia

Cell type Deactivation τ (msec) Desensitization τ (msec) PCa/PNa Rectification index (g+40mV/g−80mV) Peak current NP, −60 mV (pA)
Striatum
Striatal principal neurons 2.2  ± 0.1 (10) 11.5  ± 0.9 (10) 0.11  ± 0.00 (9) 0.93  ± 0.02 (9) 692  ± 95 (9)
Striatal cholinergic interneurons 1.2  ± 0.1 (10) 3.6  ± 0.4 (10) 1.16  ± 0.16 (10) 0.49  ± 0.06 (10) 246  ± 18 (10)
Substantia nigra
Nigral GABAergic neurons 1.7  ± 0.2 (8) 3.6  ± 0.3 (9) 0.11  ± 0.01 (8) 1.08  ± 0.17 (7) 326  ± 46 (7)
Nigral dopaminergic neurons 1.6  ± 0.1 (8) 6.1  ± 0.8 (9) 0.10  ± 0.01 (6) 1.01  ± 0.07 (7) 571  ± 90 (7)
Globus pallidus and subthalamic nucleus
Globus pallidus neurons 1.1  ± 0.1 (5) 5.1  ± 0.8 (5) 0.67  ± 0.14 (7) 0.70  ± 0.06 (7) 166  ± 60 (7)
Subthalamic nucleus neurons 1.6  ± 0.1 (8) 3.6  ± 0.4 (9) 1.17  ± 0.30 (6) 0.60  ± 0.06 (6) 285  ± 60 (6)

Deactivation and desensitization time constants of AMPARs were determined using 1 and 100 msec pulses of glutamate, respectively. ThePCa/PNa of AMPARs was calculated from the shift of reversal potential of glutamate-activated currents on exchange of Na+-rich by Ca2+-rich (30 mm) external solution. The rectification index of the AMPAR-mediated current,g+40mV/g−80mV, was determined in Na+-rich external solution. The peak current amplitude at −60 mV was determined in Na+-rich external solution. All data were obtained from nucleated patches (NP) using 3 mm glutamate, with the exception of AMPAR kinetics, which was studied in outside-out patches using 1 mm glutamate.