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. 1997 Oct 15;17(20):7796–7804. doi: 10.1523/JNEUROSCI.17-20-07796.1997

Table 1.

CaP axon morphology after muscle pioneer ablation

Immunolabel experimental (24 h) Intracellular label experimental (30 h) Intracellular label control (30 h)
n (%) n (%) n (%)
Normal axon1-a 47 84 13 100 1181-b 100
Short axon1-c 9 16 0 0 0 0
Total CaPs examined 56 100 13 100 118 100
F1-a

 No obvious difference in length or pathway choice between experimental and control CaPs, although some had abnormal branches (see Fig. 6).

F1-b

 Data from Eisen et al. (1986), 64 CaPs; (1990), 28 CaPs; Liu and Westerfield (1990), 26 CaPs.

F1-c

 CaP axons in experimental segments were determined to be short by comparing them with axons in adjacent control segments. Although CaP axons do not all grow at the same rate (Eisen et al., 1986), the large normalized length disparities seen here (experimental segments, 0.10 ± 0.12; control segments 0.64 ± 0.23; average ±SD; n = 9) were confined to segments in which the muscle pioneers were ablated.