Table 1.
CaP axon morphology after muscle pioneer ablation
| Immunolabel experimental (24 h) | Intracellular label experimental (30 h) | Intracellular label control (30 h) | ||||
|---|---|---|---|---|---|---|
| n | (%) | n | (%) | n | (%) | |
| Normal axon1-a | 47 | 84 | 13 | 100 | 1181-b | 100 |
| Short axon1-c | 9 | 16 | 0 | 0 | 0 | 0 |
| Total CaPs examined | 56 | 100 | 13 | 100 | 118 | 100 |
No obvious difference in length or pathway choice between experimental and control CaPs, although some had abnormal branches (see Fig. 6).
Data from Eisen et al. (1986), 64 CaPs; (1990), 28 CaPs; Liu and Westerfield (1990), 26 CaPs.
CaP axons in experimental segments were determined to be short by comparing them with axons in adjacent control segments. Although CaP axons do not all grow at the same rate (Eisen et al., 1986), the large normalized length disparities seen here (experimental segments, 0.10 ± 0.12; control segments 0.64 ± 0.23; average ±SD; n = 9) were confined to segments in which the muscle pioneers were ablated.