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. 1997 Oct 15;17(20):7583–7593. doi: 10.1523/JNEUROSCI.17-20-07583.1997

Fig. 5.

Fig. 5.

The repressive element can account for the majority of the tissue specificity of the 386 bp GAP-43 promoter.A, Mutations in sites A andC, which were defined in Figure 4, were introduced into the 386 bp GAP-43 promoter. B, The GAP-43 promoters with the indicated mutations were tested for activity in neurons and hepatoma cells (same as Fig. 1). C, The ratio of activity in neurons to activity in hepatoma cells is reported for each promoter construct.

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