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. 2019 Sep 3;47:518–528. doi: 10.1016/j.ebiom.2019.08.051

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© 2019 Published by Elsevier B.V.

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 3 — PCAs of the lipidomics dataset in amyloid (+) and amyloid (−) asymptomatic individuals at-risk for AD. a: Distribution of males and females according to the two principal components (females in hollow triangles, males in filled in triangles); b: Distribution of APOEε4 carriers and APOEε4 non carriers according to the two principal components (APOEε4 negative in hollow squares, APOEε4 positive in filled in squares); c: Distribution of amyloid (+) and amyloid (−) subjects according to the two principal components (hollow circles for amyloid (−) filled in circles for amyloid (+) after correction for APOE genotype and sex.