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. 2019 Oct 11;10:1135. doi: 10.3389/fphar.2019.01135

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Copyright © 2019 Wen, Zhang, Liu, Wang, Li, Yang, Wang, Cai, Li and Zhao

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Pretreatment with SAL could attenuate the inhibition effect of DOX in mitochondrial respiration. Mitochondrial respiration was measured by a Seahorse XFp apparatus detecting the basal OCR and ECAR. (A) OCR. (B) ECAR. (C) Baseline OCR. (D) Baseline ECAR. (E) Stressed OCR. (F) Stressed ECAR. ^** P < 0.01, versus control group; ^## P < 0.01, versus DOX group. All data are presented as mean ± SD (n = 3). SAL, salsolinol; DOX, doxorubicin; OCR, oxygen consumption rate; ECAR, extracellular acidification rate.