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. 2019 Aug 30;4:33. doi: 10.1038/s41392-019-0068-3

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Therapeutic effects of EGCG/AA NPs in the APP/PS1 mouse model of AD. a GFAP and ThS immunostaining indicate the status of neuroinflammation and Aβ plaque deposition, respectively. Bar diagrams demonstrate the reduction in Aβ plaque deposition at the b hippocampus and c cortex area. d Percentage cumulative drug release showed sustained drug release of EGCG/AA NPs over free EGCG. e Schematic diagram of therapeutic effects of EGCG/AA NPs in an AD mouse model. Data are expressed as the mean ± SEM *P < 0.01, **P < 0.01, ***P < 0.001, ****P < 0.0001. Reproduced from ref. ¹¹⁴